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[其他康复] 国外康复医学新进展(原文翻译)-- 髌股关节疼痛综合征

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发表于 2007-5-10 21:07:37 | 显示全部楼层 |阅读模式
髌股关节疼痛综合征是康复中心非常常见的病案,但是目前一些公认的方案不能解决问题,许多康复医生和物理治疗师发现髌股关节疼痛综合征既难诊断又难治疗。现将2007年1月15日发表在《美国内科医生》杂志上的一篇关于髌股关节疼痛综合征的最新文献报道和大家一起分享。

Management of Patellofemoral Pain Syndrome

SAMEER DIXIT, M.D., and JOHN P. DIFIORI, M.D., University of California, Los Angeles, Los Angeles, California

MONIQUE BURTON, M.D., University of Washington, Seattle, Washington

BRANDON MINES, M.D., Emory
   University, Atlanta, Georgia

Definition

The term "PFPS" is often used interchangeably with "anterior knee pain" or "runner's knee." PFPS can be defined as anterior knee pain involving the patella and retinaculum that excludes other intraarticular and peripatellar pathology.5 Chondromalacia patellae, a condition in which there is softening of the patellar articular cartilage, occurs in only a subset of patients who present with anterior knee pain.5-7

Anatomy and Biomechanics

The patellofemoral joint comprises the patella and the femoral trochlea. The patella acts as a lever and also increases the moment arm of the patellofemoral joint, the quadriceps and patellar tendons.8 Contact of the patella with the femur is initiated at 20 degrees of flexion and increases with further knee flexion, reaching a maximum at 90 degrees.9
  

Stability of the patellofemoral joint involves dynamic and static stabilizers (Figure 1), which control movement of the patella within the trochlea, referred to as "patellar tracking." Patellar tracking can be altered by imbalances in these stabilizing forces affecting the distribution of forces along the patellofemoral articular surface, the patellar and quadriceps tendons, and the adjacent soft tissues. Forces on the patella range from between one third and one half of a person's body weight during walking to three times body weight during stair climbing and up to seven times body weight during squatting.10 Abnormalities of patellar tracking must be understood to appreciate the possible causes of PFPS and to determine the focus of treatment.

Risk Factors

Several factors may create a predisposition for the development of PFPS via alterations in patellar tracking, increased patellofemoral joint forces, or combinations of these biomechanical features (Table 1).7,11-15 Overuse, trauma, and anatomic factors appear to be the main contributors.

Lower extremity malalignment (caused by abnormalities such as an increased standing Q angle, pes planus, or subtalar pronation) often has been implicated as a cause of PFPS. However, evidence to support a causal relationship between static measures of lower extremity malalignment and lower extremity injury is limited.11,16,17 In one prospective study, a small subgroup of runners with PFPS was found to have differences in ankle dorsiflexion, genu varum, and forefoot varus compared with noninjured participants.11 Analyses that include a dynamic component may eventually yield more useful information on the role of lower extremity morphology in the development of PFPS.11

Diagnosis

The differential diagnosis of PFPS is summarized in Table 2.12 For most patients, a careful history and physical examination are sufficient to make the diagnosis of PFPS.

history

Patients with PFPS typically describe pain "behind," "underneath," or "around" the patella. The symptoms are usually of gradual onset, although some cases can be caused by trauma, and may be bilateral. Common symptoms include stiffness or pain, or both, on prolonged sitting with the knees flexed (sometimes called the "theater sign"), and pain with activities that load the patellofemoral joint, such as climbing or descending stairs, squatting, or running. The pain can be difficult for the patient to localize. If asked to point to the location of pain, patients may place their hands over the anterior aspect of the knee or draw a circle with their fingers around the patella (the "circle sign"). The pain usually is described as "achy," but it can be sharp at times.

Patients may complain of the knee giving way. This usually does not represent true patellar instability but rather transient inhibition of the quadriceps because of pain or deconditioning.13 However, it is important to determine whether patellar subluxation or dislocation has occurred, because patellar instability can be associated with PFPS.

Swelling of the knee is not characteristic of PFPS, although patients may report a sensation of stiffness, especially when the knee is flexed. A "popping" or "catching" sensation may be described. Locking of the joint is not a symptom of PFPS and suggests a meniscal tear or loose bodies.

Because PFPS often is related to overuse, recent changes in activities and any changes in the frequency, duration, and intensity of training should be noted. Other possible contributors include inappropriate or excessively worn footwear, and lower extremity resistance training and conditioning activities (particularly squats and lunges). A history of injuries, including patellar subluxation or dislocation, trauma, or surgeries, should be noted because they may cause direct injury to the articular cartilage or alter the forces across the patellofemoral joint, resulting in anterior knee pain.

physical examination

A complete examination of the knee, including a careful assessment of the patellofemoral joint, should be performed (Table 312,13). The examination should aim to identify features that may alter patellofemoral mechanics.

Inspection. Patients initially should be examined "from the ground up" while standing in shorts. Although the clinical utility of static measurements of lower extremity alignment appears to be limited, such measurements may be performed at this point in the examination. Observation of the patient's gait may reveal excessive subtalar pronation.

Dynamic patellar tracking can be assessed by having the patient perform a single leg squat and stand. Imbalance between the medial and lateral patellar forces (caused by vastus medialis obliquus [VMO] dysfunction or lateral structure tightness) can be manifested by an abrupt medial deviation of the patella as the patella engages the trochlea early in flexion, known as the "J" sign.13 Alternatively, the "J" sign may be observed with the patient supine or seated and the knee extended from a flexed position. Lateral deviation of the patella can be observed during the terminal phase of extension (Figure 2).18

Quadriceps muscle bulk, especially the VMO, should be assessed by visual inspection and comparison with the opposite side. Measurement of quadriceps muscle girth can be used as a baseline in assessing progress with rehabilitation. Any surgical scars should be noted.

Palpation. This portion of the examination should be performed with the patient supine and the knee extended. The knee should be assessed for an effusion. A joint effusion is uncommon in PFPS and should prompt evaluation for other causes of knee pain. Quadriceps muscle tone can be assessed by direct palpation at rest and with isometric contraction. Careful palpation should be performed in an attempt to isolate the location of the pain (Figure 1; Table 312,13). The ligaments also should be examined as part of the comprehensive examination.

Range of Motion. Passive and active range of motion of the knee and hip should be assessed. Pain with internal or external rotation of the hip could indicate referred pain resulting from hip joint pathology and should be evaluated further. Patients with PFPS usually demonstrate a full range of motion of the knee. Asymptomatic crepitus with range of motion is a nonspecific finding, although painful crepitus may indicate an articular cartilage injury or osteoarthritis.

Special Aspects. Clinical tests for patellar mobility and position, and provocative tests for pain should be performed. The patellar glide (Figure 314), patellar tilt (Figure 418), and patellar grind (Figure 5) tests should be performed as part of the routine assessment of patients with anterior knee pain (Table 312,13). Positive results on these tests are consistent with the diagnosis of PFPS. The patellar apprehension test is used to assess for lateral instability and is positive when pain or discomfort occurs with lateral translation of the patella.

Medial patellar instability can be assessed by displacing the patella medially with the knee extended, then flexing the knee and releasing the patella. Pain indicates medial subluxation.14 Finally, flexibility of the iliotibial band (ITB), quadriceps, hamstrings, hip flexors, and the gastrocnemius should be evaluated. Tightness of the ITB and tightness of the quadriceps have been shown to be risk factors for PFPS.15,19 Poor flexibility in these areas may contribute to stress across the patellofemoral joint, and attention should be directed to this in therapy.

Imaging

PFPS is primarily a clinical diagnosis and, for many patients, treatment can be initiated without imaging. Radiography is an adjunct to the history and physical examination and should be performed in patients with a history of trauma or surgery, those with an effusion, and those whose pain does not improve with treatment. Radiography also may be helpful if a symptomatic bipartite patella is suspected.

In persons older than 50 years, radiography should be considered to assess for patellofemoral osteoarthritis. In patients who are skeletally immature, radiography may be helpful to evaluate for other causes of anterior knee pain, such as osteochondritis dissecans, physeal injury, or bone tumors. Other radiographic findings that may mimic PFPS include loose bodies and occult fractures.

When indicated, radiography should include the following views: weight-bearing anterior-posterior, weight-bearing true lateral, and axial. The axial view is taken with 20 to 45 degrees of knee flexion. Abnormalities of lateral patellar displacement, lateral patellar tilt, and dysplasia of the trochlea can be assessed on the axial view. Although these findings may indicate malalignment, they also may be seen in asymptomatic patients and are not predictive of outcome.20,21 In symptomatic patients, such findings, in combination with the physical examination, may assist in tailoring treatment.

Computed tomography and magnetic resonance imaging (MRI) are not necessary for most patients with PFPS. MRI can be helpful in detecting articular cartilage injuries, chondromalacia patellae, patellar stress fractures, and loose bodies. In addition, a pattern of marrow edema involving the medial aspect of the patella and the lateral aspect of the femoral condyle, and tears of the patellofemoral ligament can be seen with MRI and are suggestive of patellar subluxation or dislocation.

Treatment

The management of PFPS should focus on the implementation of a comprehensive rehabilitation program. It is important to communicate to the patient that a successful return to recreational or competitive activities requires compliance with the rehabilitation plan.

relative rest

Reduction of loading to the patellofemoral joint and surrounding soft tissues is the first step to reduce pain. If resistance training exercises have been identified as playing a role in causing the injury, cessation of specific exercises such as full squats and lunges is indicated. Runners should reduce mileage to a level that does not provoke pain (while running or the day after running). Alternative activities such as bicycling, swimming, or the use of an elliptical trainer can be used to maintain fitness while treatment is ongoing. Ice or other methods of cold application may further reduce symptoms. Heat is generally not recommended. Patients may find ice application after activity particularly helpful. Although symptoms usually abate with these methods, further treatment is necessary to avoid recurrence.

physical therapy

A well-structured rehabilitation program is the mainstay of treatment. Several studies have shown physical therapy to be effective in treating PFPS.21-24 However, there is no one program that will be effective for all patients. The rehabilitation program should focus on correcting maltracking of the patella by addressing the findings identified on the physical examination. Some patients may require significant strengthening of the quadriceps. Others may have excellent quadriceps strength but excessively tight lateral structures or poor quadriceps flexibility. Soft tissue techniques and flexibility exercises can be helpful for these patients. A detailed assessment of the imbalances of patellar tracking is therefore essential to tailoring treatment. Specific exercises can then be prescribed as part of a home rehabilitation program. Patients who require further assessment or ongoing instruction can be referred to a physical therapist.

addressing the underlying cause

In most patients with PFPS, a careful history will identify a precipitating event. Changes in activity patterns, such as an increase in running mileage, running stadium steps for conditioning, or the addition of resistance training exercises that affect the patellofemoral joint, often are associated with symptom onset. Excessively worn or inappropriate footwear also may contribute. Discussing these issues with the patient and developing a specific plan to avoid repeating the causal behavior are important in preventing a recurrence.

other treatments

Analgesics. Although nonsteroidal anti-inflammatory drugs (NSAIDs) are commonly prescribed for patients with PFPS, there is little evidence supporting their effectiveness.25 NSAIDs or acetaminophen may be considered at the initiation of treatment for patients with symptoms during daily activities and for those whose symptoms are not controlled with ice applications.

Bracing. A variety of braces, sleeves, and straps have been used in the treatment of PFPS. Although bracing alone may provide some symptomatic relief, three prospective randomized studies found no significant benefit when bracing was used in addition to physical therapy.26-28

Patellar Taping. Patellar taping has been suggested as a method to treat PFPS by improving alignment and quadriceps function. Although the results from uncontrolled studies were encouraging, the results of three randomized clinical trials have not been consistent: two found no benefit when patellar taping was added to a program of physical therapy.29-31 More studies are needed to determine the role of patellar taping in treating PFPS.

Foot Orthoses. As discussed above, prospective studies have yet to demonstrate strong relationships between static measures of lower extremity malalignment and lower extremity injuries. Moreover, alignment was not found to be predictive of outcome in patients with PFPS in two long-term studies.32,33 Other studies, however, have shown that orthoses can be effective in some patients with PFPS.22,34 Over-the-counter soft orthoses are a reasonable choice for patients who have PFPS with malalignment. For those with persistent symptoms, a custom orthotic can be considered.

Long-term Outcomes

There are few long-term studies on the treatment of PFPS. Two studies in which patients were instructed on a program of home exercises reported successful outcomes in approximately 75 to 85 percent of patients with PFPS.33,35 A study of athletes who visited a sports medicine clinic and were instructed on VMO training found that 54 percent were pain free or had mild symptoms after nearly six years.36 Interestingly, arthroscopy findings of the patellar articular surface have not been shown to be predictive of outcome.33 Findings associated with a poorer result include a hypermobile patella, older age, bilateral symptoms, and patellar pain and crepitation on examination.35,36

Surgical Consultation

Surgical consultation for PFPS may be considered for those patients whose symptoms persist despite their completing at least six to 12 months of a thorough program of rehabilitation, and in whom other causes of anterior knee pain have been excluded.

Most studies of surgical treatment for patellofemoral disorders are uncontrolled case series. Controlled studies of surgical outcomes are limited and are highly dependent on proper patient selection. Because it is essential that the surgical procedure specifically address the individual characteristics of patellar maltracking in each patient, consultation should be obtained from a surgeon with significant experience in treating patellofemoral joint disorders.

Surgical options include release of the lateral retinaculum, articular cartilage procedures, proximal realignment, and distal realignment-often with anteromedialization of the tibial tubercle.14 Patients with tight lateral structures may benefit from lateral release, with proximal realignment in some cases. Distal realignment with anteromedialization of the tibial tubercle may benefit those with lateral compression and associated articular cartilage injury. A full discussion of the various surgical procedures and indications is beyond the scope of this review.

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 楼主| 发表于 2007-5-10 21:11:25 | 显示全部楼层

髌股关节疼痛综合征的治疗

定义

髌股关节疼痛综合征也可以称为膝前疼痛或运动员膝,膝前疼痛包括髌骨和关节内、髌周韧带的病理。髌骨软骨软化是指髌骨关节软骨的软化,只见于一部分有膝前疼痛的患者。

解剖和生物力学

髌股关节由髌骨和股骨滑车(股骨髌面)组成,髌骨就象一个杠杆,同时也可以增加髌骨关节、股四头肌和髌骨肌腱的力臂。髌骨和股骨以屈曲20度相接触,随着进一步屈膝,度数增加,最大可达到90度。

髌股关节的稳定性包括静态和动态平衡装置,它控制着髌骨和滑车中的运动,可以被称为髌骨轨道。分布于髌股关节面、髌骨和股四头肌同、肌腱力量的不平衡可以改变髌骨轨道。施加在髌骨上的力在人行走时是体重的1/31/2,在爬楼梯时是体重的3倍,在下蹲时是体重的7倍。应该懂得髌骨轨道的异常,从而理解髌股关节疼痛综合征的可能病因和治疗重点。

危险因素

这些危险因素可以影响髌股关节疼痛综合征的发展,包括改变髌骨轨道、增加髌股关节压力或是这些生物力学因素的结合,过度使用,外伤,解剖因素可能是主要的原因。

下肢对线不齐(?malalignment)通常被认为是髌股关节疼痛综合征的病因之一,但是下肢对线不齐的静态测量和下肢损伤之间因果联系的证据有限。在一个前瞻性研究中,有髌股关节疼痛综合征的运动员组和无外伤的对照组相比,在踝背屈、膝内翻、足内翻。对动态结构的分析能为下肢形态学在髌股关节疼痛综合征的发展中所起的作用提供了有用的信息。

诊断

髌股关节疼痛综合征的鉴别诊断归纳在表2.12中,应对大多数患者进行详细的病史采集和体格检查来明确髌股关节疼痛综合征的诊断。

有髌股关节疼痛综合征的患者典型的疼痛位于髌骨后面、下面或周围。症状通常逐渐发生,有一些病例可以是外伤引起,可以为双侧。常见的症状包括僵硬、疼痛、或两者都有,出现在长时间的屈膝坐,上下楼梯、下蹲或跑步等对髌股关节施加压力的活动中。患者可能难于准确定位疼痛。如果要患者指出疼痛的部位,他们可能会把他们的手放在膝盖前面或用手指在髌骨上画个圈。疼痛通常为钝痛,有时可能为锐痛。

患者可能会说他们的膝关节坏了,这通常不能代表髌骨不稳,而是四头肌因为疼痛出现的短暂抑制。然而,我们应该重视髌骨是否有半脱位或脱位,因为髌骨不稳可以和髌股关节疼痛综合征一起发生。

膝关节肿胀不是髌股关节疼痛综合征的一个特征,尽管患者可能会描述为一种僵硬感,特别是屈膝时。患者可能会描述爆音感或绊住感。关节绞锁不是髌股关节疼痛综合征的症状之一,它仅提示半月板撕裂或松驰。

因为髌股关节疼痛综合征通常与过度使用有关,所以最近活动的改变,任何训练频率、持续时间、强度的改变应该被重视。其他可能的因素包括不适合或用旧的鞋,下肢抗阻训练和蹲和跳跃训练。应该注意外伤史,如髌骨半脱位或脱位,损伤或手术,因为他们可能引起关节软骨的直接损伤或改变髌股关节上的压力,从而导致膝前疼痛。

体格检查

应该进行详细的膝关节检查,包括对髌股关节详细的评定。检查应以发现改变髌股关节力学特征为准。

视诊

患者最初应穿短裤站立位被详细的检查。尽管对下肢对线的静态测量的临床应用可能会受到限制,这些测量仍然应该在这个时候进行。观察患者的步态可以揭示距下旋后(subtalar pronation)。

对髌骨轨道的动态检查应该在患者单腿蹲和站立时进行。髌骨内外压力不平衡可以通过髌骨在滑车中早期屈曲时突然向内偏表现出来,也就是J征。J征也可以让患者仰卧或坐位时屈膝时伸膝观察。髌骨向外侧偏可以在伸膝的终末相观察。股四头肌特别是股内侧肌应该被视诊并和对侧比较。股四头肌周径可以做为康复过程中评定的基线。应注意外科伤疤。

触诊

这个检查应该在患者仰卧伸膝时进行。应观察膝有没有渗出。渗出在髌股关节疼痛综合征中并不常见,所以如果有的话应考虑是否有其他原因引起的膝痛。股四头肌的张力可以在静息时和等长收缩时直接触诊。应该仔细触诊明确疼痛定位。也应该检查韧带做为全面检查的一部分。

关节活动度

应该评定膝和髋的主动和被动活动度。髋内旋或外旋时出现的疼痛提示髋关节病理的牵涉痛,应作进一步评定。髌股关节疼痛综合征的患者膝关节活动度通常正常。膝关节活动时出现的无症状的关节响声没有特异性,有疼痛的关节响声可能提示关节软骨损伤或骨关节炎。

特殊方面

应该进行髌骨活动性和位置的临床检查和激发疼痛的检查。髌骨滑动、髌骨倾斜和髌骨研磨检查应做为膝前商务通的常规检查。这些检查的阳性结果提示髌股关节疼痛综合征的诊断。髌骨疼痛检查 被用在主人外侧不逢君之恶性,当髌骨向外时出现疼痛或不适。

髌骨内侧不稳可以在髌骨伸膝位向内移然后屈膝放开髌骨时评定。最后,应评定髂胫束、股四头肌、国绳肌、屈髋肌和腓肠肌的伸展性。髂胫束和股四头肌的紧张度可以作为髌股关节疼痛综合征的危险因素。这些部位伸展性差可以对髌股关节施加压力,在治疗时应该重视。

影像学

 髌股关节疼痛综合征的诊断是临床诊断,对大多数患者来说,可以不用影像学资料就做出治疗。X线检查是对病史和体格检查的补充,应该在有外伤史或手术史的患者、有渗出的和疼痛不随治疗好转的患者身上进行。X线检查也有助于诊断有症状的双髌骨。

对于年龄超过50岁的患者,应该进行X线检查排除是否有髌股关节骨关节炎。对于骨骼未成熟的患者,X线检查有助于评价其他原因引起的膝前痛,如骨软骨炎,骺损伤,骨肿瘤。其他象髌股关节疼痛综合征的影像学发现还有松驰小体和隐匿性骨折。

如果要进行影像学检查,应包括负重前后位,负重真侧位和轴位。轴位是膝屈曲20到45度。轴位上可以观察到髌骨外侧移位,髌骨外侧倾斜和滑车发育不良等异常。尽管这些发现提示对线不齐,但它们也见于无症状的患者,所以不能预测结果。对于有症状的患者,这些发现连同体格检查可以帮助制定治疗方案。CT和MRI对于大多数患者来说并不必要。MRI可以用于发现关节软骨损伤,髌骨软化,髌骨应力性骨折和松驰小体。另外,髌骨内侧面和股骨髁外侧面的骨髓水肿和髌股韧带的撕裂在MRI上被发现,常提示髌骨半脱位或脱位。

治疗 

髌股关节疼痛综合征的治疗应着重于全面的康复治疗。让患者明白成功恢复娱乐和竞技活动需要依从于康复治疗。

减少髌股关节和周围软组织的压力是减轻疼痛的第一步。如果抗阻训练在引起损伤中起作用,那么应停止象全蹲和跳跃这类特殊训练。运动员应该减少每天跑步量至不引起第二天疼痛的水平。骑自行车、游泳和运用椭圆形的运动员可以在治疗时同时进行这些运动。冰和其他冷疗可以进一步减轻症状。不推荐用热疗。患者可以发现运动后用冰特别有效。尽管这些方法可以减轻症状,但应有进一步治疗以避免复发。

物理治疗 

设计完好的康复治疗计划是治疗的主体。有一些研究证明物理治疗对髌股关节疼痛综合征有效。康复治疗应着重通过体格检查发现纠正髌骨错误的轨道。有一些患者可能需要着重增加股四头肌肌力。其他的患者可能有比较好的股四头肌肌力,但可能外侧结构太紧或股四头肌伸展性差。对于这类患者,软组织技术和伸展训练应有帮助。详细评定髌骨轨道的不平衡对制定治疗计划是非常基本的。特殊的训练可以作为家庭康复治疗的一部分。需要进一步评定或治疗指导的患者可以去找物理治疗师。

寻找潜在的病因

对于大多数髌股关节疼痛综合征的患者而言,在详细的病史采集中应明确有没有突发事件。在运动模式上的改变,如增加跑步英里数,跑训练运动声的台阶,增加影响髌股关节的抗阻训练,通常与症状发生有关。过度磨损或不合适的鞋也可以起作用。与患者讨论这些问题,制定个体化的治疗可以避免重复这些损伤行为,对预防复发很重要。

其他治疗

止痛药

NSAIDS通常开给有髌股关节疼痛综合征的患者,但极少有证据支持它们的有效性。NSAIDS或对乙酰氨基酚可以用在日常活动中有症状或冷疗无效的患者的最初治疗中。

矫形器

矫形器、袖子、带子已经应用于髌股关节疼痛综合征的治疗中。尽管单独使用矫形器能缓解一部分症状,但有3个前瞻性对照研究发现矫形器结合物理治疗无效。

髌骨绷带

髌骨绷带已经作为髌股关节疼痛综合征治疗中改善对线和股四头肌功能的方法。尽管无对照研究的结果证明有效,但有3个随机临床试验的结果并不一致,有2个试验证明髌骨绷带结合物理治疗无效。需要进行更多的试验来证明髌骨绷带在治疗髌股关节疼痛综合征的作用。

足矫形器

正如前面所讨论的,需要进行前瞻性研究来论证下肢对线的静态测量和下肢损伤之间的关系。有2个长期的研究发现对线不能作为髌股关节疼痛综合征患者结局的预测。另外有一研究发现矫形器对某些髌股关节疼痛综合征的患者有效。非处方的软性矫形器可以作为对线不齐的髌股关节疼痛综合征患者的选择。这些有持续症状的患者,应该使用定制的矫形器。

远期结局

关于髌股关节疼痛综合征治疗的远期研究很少。有2个研究发现有70-85%髌股关节疼痛综合征的患者在家庭康复治疗中得到好的结果。有一个关于运动员在运动医学治疗所治疗,进行股四头肌内侧群的训练,有54%的患者在治疗6年后疼痛消失或减轻到很小。有趣的是,关节镜检查发现髌骨关节表面不能作为结果的预测。研究发现,髌骨活动性高、高龄、有双侧症状和髌骨疼痛、检查时有响声有不好的结局。

外科咨询

外科咨询应该用在那些症状在全面康复治疗6至12个月不缓解和由其他原因引起的膝前疼痛的患者中。

大多数有关髌股关节紊乱的手术治疗研究都是无对照的病例系列研究。手术治疗的对照研究很受限,并高度依赖于病例的正确选择。因为外科治疗过程特别强调髌骨错误轨道的个体特征,所以在治疗髌股关节紊乱方面有经验的外科医生应详细咨询。

外科过程包括了外侧韧带的可松解,关节软骨过程,近端重排和远端重排。外侧结构太紧的患者可以从外侧结构松解,近端重排中受益。胫骨结节前内移位的远端重排对有外侧压迫合并关节软骨损伤的患者有用。这篇文章不会详细讨论不同外科治疗过程和指征。

感谢 kelly bag 为此文翻译作出的贡献!

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 楼主| 发表于 2007-5-10 21:12:53 | 显示全部楼层

髌股关节疼痛综合征的治疗

定义

髌股关节疼痛综合征也可以称为膝前疼痛或运动员膝,膝前疼痛包括髌骨和关节内、髌周韧带的病理。髌骨软骨软化是指髌骨关节软骨的软化,只见于一部分有膝前疼痛的患者。

解剖和生物力学

髌股关节由髌骨和股骨滑车(股骨髌面)组成,髌骨就象一个杠杆,同时也可以增加髌骨关节、股四头肌和髌骨肌腱的力臂。髌骨和股骨以屈曲20度相接触,随着进一步屈膝,度数增加,最大可达到90度。

髌股关节的稳定性包括静态和动态平衡装置,它控制着髌骨和滑车中的运动,可以被称为髌骨轨道。分布于髌股关节面、髌骨和股四头肌同、肌腱力量的不平衡可以改变髌骨轨道。施加在髌骨上的力在人行走时是体重的1/31/2,在爬楼梯时是体重的3倍,在下蹲时是体重的7倍。应该懂得髌骨轨道的异常,从而理解髌股关节疼痛综合征的可能病因和治疗重点。

危险因素

这些危险因素可以影响髌股关节疼痛综合征的发展,包括改变髌骨轨道、增加髌股关节压力或是这些生物力学因素的结合,过度使用,外伤,解剖因素可能是主要的原因。

下肢对线不齐(?malalignment)通常被认为是髌股关节疼痛综合征的病因之一,但是下肢对线不齐的静态测量和下肢损伤之间因果联系的证据有限。在一个前瞻性研究中,有髌股关节疼痛综合征的运动员组和无外伤的对照组相比,在踝背屈、膝内翻、足内翻。对动态结构的分析能为下肢形态学在髌股关节疼痛综合征的发展中所起的作用提供了有用的信息。

诊断

髌股关节疼痛综合征的鉴别诊断归纳在表2.12中,应对大多数患者进行详细的病史采集和体格检查来明确髌股关节疼痛综合征的诊断。

有髌股关节疼痛综合征的患者典型的疼痛位于髌骨后面、下面或周围。症状通常逐渐发生,有一些病例可以是外伤引起,可以为双侧。常见的症状包括僵硬、疼痛、或两者都有,出现在长时间的屈膝坐,上下楼梯、下蹲或跑步等对髌股关节施加压力的活动中。患者可能难于准确定位疼痛。如果要患者指出疼痛的部位,他们可能会把他们的手放在膝盖前面或用手指在髌骨上画个圈。疼痛通常为钝痛,有时可能为锐痛。

患者可能会说他们的膝关节坏了,这通常不能代表髌骨不稳,而是四头肌因为疼痛出现的短暂抑制。然而,我们应该重视髌骨是否有半脱位或脱位,因为髌骨不稳可以和髌股关节疼痛综合征一起发生。

膝关节肿胀不是髌股关节疼痛综合征的一个特征,尽管患者可能会描述为一种僵硬感,特别是屈膝时。患者可能会描述爆音感或绊住感。关节绞锁不是髌股关节疼痛综合征的症状之一,它仅提示半月板撕裂或松驰。

因为髌股关节疼痛综合征通常与过度使用有关,所以最近活动的改变,任何训练频率、持续时间、强度的改变应该被重视。其他可能的因素包括不适合或用旧的鞋,下肢抗阻训练和蹲和跳跃训练。应该注意外伤史,如髌骨半脱位或脱位,损伤或手术,因为他们可能引起关节软骨的直接损伤或改变髌股关节上的压力,从而导致膝前疼痛。

体格检查

应该进行详细的膝关节检查,包括对髌股关节详细的评定。检查应以发现改变髌股关节力学特征为准。

视诊

患者最初应穿短裤站立位被详细的检查。尽管对下肢对线的静态测量的临床应用可能会受到限制,这些测量仍然应该在这个时候进行。观察患者的步态可以揭示距下旋后(subtalar pronation)。

对髌骨轨道的动态检查应该在患者单腿蹲和站立时进行。髌骨内外压力不平衡可以通过髌骨在滑车中早期屈曲时突然向内偏表现出来,也就是J征。J征也可以让患者仰卧或坐位时屈膝时伸膝观察。髌骨向外侧偏可以在伸膝的终末相观察。股四头肌特别是股内侧肌应该被视诊并和对侧比较。股四头肌周径可以做为康复过程中评定的基线。应注意外科伤疤。

触诊

这个检查应该在患者仰卧伸膝时进行。应观察膝有没有渗出。渗出在髌股关节疼痛综合征中并不常见,所以如果有的话应考虑是否有其他原因引起的膝痛。股四头肌的张力可以在静息时和等长收缩时直接触诊。应该仔细触诊明确疼痛定位。也应该检查韧带做为全面检查的一部分。

关节活动度

应该评定膝和髋的主动和被动活动度。髋内旋或外旋时出现的疼痛提示髋关节病理的牵涉痛,应作进一步评定。髌股关节疼痛综合征的患者膝关节活动度通常正常。膝关节活动时出现的无症状的关节响声没有特异性,有疼痛的关节响声可能提示关节软骨损伤或骨关节炎。

特殊方面

应该进行髌骨活动性和位置的临床检查和激发疼痛的检查。髌骨滑动、髌骨倾斜和髌骨研磨检查应做为膝前商务通的常规检查。这些检查的阳性结果提示髌股关节疼痛综合征的诊断。髌骨疼痛检查 被用在主人外侧不逢君之恶性,当髌骨向外时出现疼痛或不适。

髌骨内侧不稳可以在髌骨伸膝位向内移然后屈膝放开髌骨时评定。最后,应评定髂胫束、股四头肌、国绳肌、屈髋肌和腓肠肌的伸展性。髂胫束和股四头肌的紧张度可以作为髌股关节疼痛综合征的危险因素。这些部位伸展性差可以对髌股关节施加压力,在治疗时应该重视。

影像学

 髌股关节疼痛综合征的诊断是临床诊断,对大多数患者来说,可以不用影像学资料就做出治疗。X线检查是对病史和体格检查的补充,应该在有外伤史或手术史的患者、有渗出的和疼痛不随治疗好转的患者身上进行。X线检查也有助于诊断有症状的双髌骨。

对于年龄超过50岁的患者,应该进行X线检查排除是否有髌股关节骨关节炎。对于骨骼未成熟的患者,X线检查有助于评价其他原因引起的膝前痛,如骨软骨炎,骺损伤,骨肿瘤。其他象髌股关节疼痛综合征的影像学发现还有松驰小体和隐匿性骨折。

如果要进行影像学检查,应包括负重前后位,负重真侧位和轴位。轴位是膝屈曲20到45度。轴位上可以观察到髌骨外侧移位,髌骨外侧倾斜和滑车发育不良等异常。尽管这些发现提示对线不齐,但它们也见于无症状的患者,所以不能预测结果。对于有症状的患者,这些发现连同体格检查可以帮助制定治疗方案。CT和MRI对于大多数患者来说并不必要。MRI可以用于发现关节软骨损伤,髌骨软化,髌骨应力性骨折和松驰小体。另外,髌骨内侧面和股骨髁外侧面的骨髓水肿和髌股韧带的撕裂在MRI上被发现,常提示髌骨半脱位或脱位。

治疗 

髌股关节疼痛综合征的治疗应着重于全面的康复治疗。让患者明白成功恢复娱乐和竞技活动需要依从于康复治疗。

减少髌股关节和周围软组织的压力是减轻疼痛的第一步。如果抗阻训练在引起损伤中起作用,那么应停止象全蹲和跳跃这类特殊训练。运动员应该减少每天跑步量至不引起第二天疼痛的水平。骑自行车、游泳和运用椭圆形的运动员可以在治疗时同时进行这些运动。冰和其他冷疗可以进一步减轻症状。不推荐用热疗。患者可以发现运动后用冰特别有效。尽管这些方法可以减轻症状,但应有进一步治疗以避免复发。

物理治疗 

设计完好的康复治疗计划是治疗的主体。有一些研究证明物理治疗对髌股关节疼痛综合征有效。康复治疗应着重通过体格检查发现纠正髌骨错误的轨道。有一些患者可能需要着重增加股四头肌肌力。其他的患者可能有比较好的股四头肌肌力,但可能外侧结构太紧或股四头肌伸展性差。对于这类患者,软组织技术和伸展训练应有帮助。详细评定髌骨轨道的不平衡对制定治疗计划是非常基本的。特殊的训练可以作为家庭康复治疗的一部分。需要进一步评定或治疗指导的患者可以去找物理治疗师。

寻找潜在的病因

对于大多数髌股关节疼痛综合征的患者而言,在详细的病史采集中应明确有没有突发事件。在运动模式上的改变,如增加跑步英里数,跑训练运动声的台阶,增加影响髌股关节的抗阻训练,通常与症状发生有关。过度磨损或不合适的鞋也可以起作用。与患者讨论这些问题,制定个体化的治疗可以避免重复这些损伤行为,对预防复发很重要。

其他治疗

止痛药

NSAIDS通常开给有髌股关节疼痛综合征的患者,但极少有证据支持它们的有效性。NSAIDS或对乙酰氨基酚可以用在日常活动中有症状或冷疗无效的患者的最初治疗中。

矫形器

矫形器、袖子、带子已经应用于髌股关节疼痛综合征的治疗中。尽管单独使用矫形器能缓解一部分症状,但有3个前瞻性对照研究发现矫形器结合物理治疗无效。

髌骨绷带

髌骨绷带已经作为髌股关节疼痛综合征治疗中改善对线和股四头肌功能的方法。尽管无对照研究的结果证明有效,但有3个随机临床试验的结果并不一致,有2个试验证明髌骨绷带结合物理治疗无效。需要进行更多的试验来证明髌骨绷带在治疗髌股关节疼痛综合征的作用。

足矫形器

正如前面所讨论的,需要进行前瞻性研究来论证下肢对线的静态测量和下肢损伤之间的关系。有2个长期的研究发现对线不能作为髌股关节疼痛综合征患者结局的预测。另外有一研究发现矫形器对某些髌股关节疼痛综合征的患者有效。非处方的软性矫形器可以作为对线不齐的髌股关节疼痛综合征患者的选择。这些有持续症状的患者,应该使用定制的矫形器。

远期结局

关于髌股关节疼痛综合征治疗的远期研究很少。有2个研究发现有70-85%髌股关节疼痛综合征的患者在家庭康复治疗中得到好的结果。有一个关于运动员在运动医学治疗所治疗,进行股四头肌内侧群的训练,有54%的患者在治疗6年后疼痛消失或减轻到很小。有趣的是,关节镜检查发现髌骨关节表面不能作为结果的预测。研究发现,髌骨活动性高、高龄、有双侧症状和髌骨疼痛、检查时有响声有不好的结局。

外科咨询

外科咨询应该用在那些症状在全面康复治疗6至12个月不缓解和由其他原因引起的膝前疼痛的患者中。

大多数有关髌股关节紊乱的手术治疗研究都是无对照的病例系列研究。手术治疗的对照研究很受限,并高度依赖于病例的正确选择。因为外科治疗过程特别强调髌骨错误轨道的个体特征,所以在治疗髌股关节紊乱方面有经验的外科医生应详细咨询。

外科过程包括了外侧韧带的可松解,关节软骨过程,近端重排和远端重排。外侧结构太紧的患者可以从外侧结构松解,近端重排中受益。胫骨结节前内移位的远端重排对有外侧压迫合并关节软骨损伤的患者有用。这篇文章不会详细讨论不同外科治疗过程和指征。

感谢 kelly bag 为此文翻译作出的贡献!

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